OSA is much more common than CSA and involves the airway being blocked by tissues in the mouth and throat. In CSA, breathing problems occur because of disrupted signaling between the brain and respiratory muscles. At Ria, insomnia after stopping drinking we offer weekly meetings with certified counselors to help members stay on track and build skills for long-term change.
What’s the Link Between Alcohol and Sleep Apnea?
Slow wave sleep increases after moderate to high bedtime alcohol use, but the effect of lower doses on slow wave sleep are https://www.fondationmichelserres.fr/understanding-alcohol-allergy-symptoms/ again less certain. Slow wave sleep effects diminish with repeated nights of alcohol consumption (13). The effects of alcohol on sleep continuity and total sleep are quite variable but appear to be dose related. Lower doses may increase total sleep time, whereas higher doses may lead to short-term withdrawal, increasing sympathetic activity and sleep disruption especially during the second half of the night.
Many of us find ourselves tossing and turning at night, trying to get that elusive 7 to 8 hours of sleep experts say we need but never finding it. Alcohol dependency is rarely the only issue a person in withdrawal is dealing with. This is why a comprehensive approach to treatment is often the key to a successful recovery. However, there are many coping skills a person can practice to improve their sleep.
Binge-drinking and Sleep
- This preliminary investigation evaluated the link between alcohol craving and insomnia in actively drinking patients with alcohol dependence (AD).
- I believe that an occasional drink may do you some good – in terms of mood, relaxation, and heart health – but overdoing it is no doubt harmful.
- This chronotype is commonly seen in adolescents and those with psychiatric disorders.
- In this population based setting, drinking high volumes of alcohol or drinking hazardously may contribute to the prevalence of sleep problems in older age.
- This typically happens to people who use alcohol to cope with the effects of mental health disorders.
- The suprachiasmatic nucleus within the hypothalamus in the brain is the master clock that synchronizes a host of internal rhythms with the sleep-wake cycle being one of them.
First, it can help to simply recognize how alcohol impacts your sleep and energy levels. Then, you can take steps—like cutting back, practicing healthy sleep hygiene, and seeking support if needed—to improve your habits and start feeling better. Alcohol craving’s association with insomnia symptoms and the insomnia global score.
Neurological Effects

Treating a co-occurring disorder without treating someone’s alcohol dependency, and vice versa, can mean setting up a possible relapse before treatment has truly begun. Rarely is it the cravings of alcohol that drive those in recovery to relapse. It is, rather, the symptoms of withdrawal taking a physical and mental toll on the person quitting that pushes them back.
Furthermore, alcohol-induced insomnia can lead to fragmented sleep patterns, reduced overall sleep time, and a lack of restorative rest. These symptoms often result in daytime fatigue, irritability, difficulty concentrating, and impaired cognitive function. It is essential to recognize these signs and address them promptly to improve sleep quality and overall well-being. Depending on the amount consumed, the habitual level of consumption, and the person’s body mass, the relaxing effects of alcohol can actually make it easier to fall asleep. A body and brain already relaxed from alcohol doesn’t need the full length of a typical Stage 1 of the sleep cycle, which transitions us from awake to sleep states. After that, however, alcohol disrupts later sleep stages as it continues to metabolize.

Weafer has expanded her research focus on risks for problem drinking to address the fact that poor sleep can lead to increased drinking and those with AUD commonly have difficulty with sleep. The ventral tegmental area (VTA) in the midbrain has numerous reciprocal inputs and projections to and from other areas of the brain. Recent studies demonstrate that neurons of the VTA may promote wakefulness, especially under conditions of high motivation (Eban-Rothschild et al., 2016; Sun et al., 2017) through their projection to the orexinergic neuron in the lateral hypothalamus.
In addition to its direct impact on sleep architecture, alcohol can also exacerbate underlying issues that contribute to insomnia, such as anxiety and depression. While it may initially make you feel drowsy and relaxed, alcohol can disrupt your body’s natural circadian rhythm and interfere with the production of melatonin, the hormone that regulates sleep-wake cycles. There have been many, many studies over the years investigating the health effects of alcohol, both positive and negative. It has been shown to raise the risk of some cancers, and it has also been shown Alcohol Use Disorder to reduce the risk of heart disease.

Alcohol Withdrawal and Sleep
A dependence on alcohol before bed doesn’t just increase sleep apnea and insomnia risk. It can also increase your risk of heart attack, stroke, arrhythmia, and even sudden death. Although alcohol can make a person feel sleepy at bedtime, it throws off the normal process of sleep. As alcohol wears off during the night, a person is more likely to have shallower, lower-quality sleep and to awaken more frequently.
Moreover, using alcohol to cope with insomnia can have negative consequences on overall health and well-being. It’s important to break this cycle and seek healthier alternatives for managing insomnia. Furthermore, alcohol can increase the occurrence of sleep fragmentation, causing frequent awakenings throughout the night. These awakenings disrupt the natural sleep cycle and can lead to a feeling of unrestfulness upon waking. Understanding the connection between alcohol and insomnia is crucial to grasp the impact that alcohol consumption can have on sleep patterns. In this section, we will explore the role of alcohol as both a sleep aid and a disruptor of sleep patterns.